Since the first comprehensive and impressive description of juvenile myoclonic epilepsy (JME) by Janz and Christian, JME has become a well-established disease entity [1]. Previous studies based on functional Magnetic Resonance Imaging (MRI), Positron Emission Tomography/ Computed Tomography (PET-CT) and scalp electroencephalogram (EEG) have illustrated at different levels that patients with JME exhibit significantly different functional and structural brain connectivity networks compared to healthy control subjects [2–5].
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