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Congenital Zika syndrome (CZS) is a severe condition caused by intrauterine exposure to the Zika virus (ZIKV). It is responsible for a 10 times higher mortality rate in children without the syndrome [1]. The phenotype for CZS varies based on neurological impairment [2,3]. It encompasses a wide spectrum of structural [4] and functional [5] alterations or sequelae in neurodevelopment that children with CZS may present in the first years of life [6].