Up to one-fourth of all cerebral cavernous malformations (CCM) affect the pediatric population [1–3]. Such lesions are a major cause of spontaneous intracerebral hemorrhage and may result in headaches, seizures, or focal neurological deficits [4]. A substantial number of lesions have a benign clinical course. However, some children suffer from CCM-related epileptic seizures (CRE), which are the most common trait of CCM disease. Pediatric CRE can lead to high morbidity, with particularly dreadful consequences in children and their development, given their long life expectancy [5–8].
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