Following the emergence of an epidemic of microcephaly associated with intrauterine Zika virus (ZIKV) infections in northeast Brazil, the Congenital Zika Syndrome (CZS) clinical phenotype has been characterized since 2016 [1,2,3,4,5,6,7,8,9,10]. Children with Zika-related microcephaly (ZRM) may present with structural abnormalities that include diffuse atrophy of the brain, cerebellum and brainstem, calcifications, and malformations of cortical development [3,11,12].
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