In patients with new-onset seizures, autoimmune limbic encephalitis (LE) is an important differential diagnosis. Consensus criteria for definite autoimmune LE require typical clinical symptoms with subacute onset (working memory deficits, seizures, or psychiatric symptoms), bilateral MRI signs of LE, and either cerebrospinal fluid (CSF) pleocytosis or focal EEG findings of the temporal lobes [1]. Detection of a typical neural autoantibody may compensate for the lack of one of these three criteria.
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