Cerebral Hyperperfusion Syndrome (CHS) is a relatively rare and disabling complication following carotid artery stenting (CAS) characterized by headache, seizures and focal neurological deficit. Radiological features include cerebral edema or intracerebral hemorrhage and it can be diagnosed with transcranial doppler (TCD), brain computed tomography (CT) or magnetic resonance imaging (MRI). Although pathophysiological mechanisms underlying CHS are still unknown, an increased regional cerebral blood flow secondary to impaired autoregulation of cerebrovascular system has been proposed [1].
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