Posterior reversible encephalopathy syndrome (PRES), as defined by Hinchey et al. (1996), is a phenomenon of transient cerebral vasogenic edema occurring preferentially in posterior circulation [1]. Clinically, PRES is characterized by headaches, seizures, reduced consciousness, and visual and other focal neurological symptoms [2,3]. PRES is a clinicoradiological syndrome; a characteristic radiologic finding is vasogenic edema in the bilateral parietal-occipital lobes, which might be related to the lower concentrations of sympathetic innervation of the posterior intracranial arteries in comparison with other cerebral regions, resulting in lower autoregulatory capacity in these vessels [4].
AGO