Since the first, impressively comprehensive description by Janz and Christian [1] juvenile myoclonic epilepsy (JME) has appeared as a well-shaped disease entity. The name-giving seizure type, indispensable for the diagnosis, was single or arrhythmic bilateral, predominantly brachial myoclonus, that could be combined with generalized tonic-clonic seizures (GTCS), mostly in the awakening phase, and absences [2]. Traditionally, JME is considered an idiopathic generalized epilepsy [2]. However, this concept is not accepted any more, but being replaced by a view focusing on a system disorder of the brain with ictogenesis in distributed, bilateral networks involving primarily the thalamus and selective areas of the neocortex [3].
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