Temporal lobe epilepsy with hippocampal sclerosis (TLE-HS) is currently understood as a disease that originates in a largely distributed neuronal network, involving temporal and extratemporal structures [1]. Neuroimaging studies have mainly placed the focus on characterizing the affected hippocampus, since surgical removal of the mesial temporal structures has proven to be effective; however, approximately 40% of patients relapse [2].
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